Why Steroid Dose Conversion Matters

Converting between corticosteroids is a routine but error-prone part of daily practice — switching a patient from IV hydrocortisone to oral prednisolone before discharge, adjusting a taper before stopping dexamethasone after a COVID-19 admission, or working out an equivalent budesonide dose for a patient with Crohn's disease who can't tolerate systemic steroids. Because corticosteroids differ enormously in anti-inflammatory potency, mineralocorticoid activity, and duration of action, a simple "double the dose" approach can leave a patient under-treated or over-exposed to unwanted sodium and fluid retention.

This steroid equivalence calculator converts between the eight corticosteroids most commonly prescribed in Indian hospitals and OPDs — hydrocortisone, cortisone acetate, prednisolone, prednisone, methylprednisolone, triamcinolone, dexamethasone, betamethasone, and oral budesonide — using published anti-inflammatory potency ratios referenced to hydrocortisone. Enter the current steroid and dose, and the tool instantly returns the equivalent dose for every other corticosteroid on the list, alongside each drug's relative mineralocorticoid activity, duration of action, and HPA-axis suppression risk.

The reference table further down expands on the anti-inflammatory potency values used, and the clinical notes below it cover common switch scenarios — prednisolone-to-hydrocortisone conversion for stress dosing, dexamethasone for cerebral oedema and antenatal lung maturity, and safe tapering to prevent adrenal insufficiency.

As with any dose-conversion tool, these equivalences are approximations drawn from anti-inflammatory potency alone — they don't account for a drug's specific indication, route of administration, or a patient's hepatic or renal function. Always cross-check with a pharmacist or senior colleague before switching steroids in a critically ill or physiologically fragile patient.

Corticosteroid Dose Converter
Enter dose in milligrams (mg)
Equivalent Doses for mg
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Corticosteroid Equivalence Reference Table

SteroidAnti-inflammatory Equiv. (mg)Anti-inflam. PotencyMineralo. PotencyDuration (hours)HPA Suppression
Cortisone acetate250.8×2+8–12hShort
Hydrocortisone (cortisol)201× (reference)2+8–12hShort
Prednisolone / Prednisone51+12–36hIntermediate
Methylprednisolone4012–36hIntermediate
Triamcinolone4012–36hIntermediate
Dexamethasone0.7525–30×036–54hLong (strong)
Betamethasone0.625–30×036–54hLong (strong)
Budesonide (oral)~1.2~15×012–24hModerate (first-pass)

Key Clinical Notes

Steroid Tapering — Preventing Adrenal Insufficiency

Frequently Asked Questions

What is steroid equivalence?
Steroid equivalence refers to the anti-inflammatory potency of different corticosteroids relative to hydrocortisone (reference = 1). Equivalent anti-inflammatory doses: hydrocortisone 20 mg = prednisolone 5 mg = methylprednisolone 4 mg = dexamethasone 0.75 mg = betamethasone 0.6 mg. Note: mineralocorticoid potency is separate — dexamethasone has negligible mineralocorticoid activity while hydrocortisone has significant fludrocortisone-like effects.
What is the difference between prednisolone and dexamethasone?
Prednisolone: moderate potency (4–5x hydrocortisone), significant mineralocorticoid activity, half-life 12–36 hours, once or twice daily dosing, first-line for most chronic inflammatory conditions. Dexamethasone: high potency (25–30x hydrocortisone), negligible mineralocorticoid activity, half-life 36–72 hours, once daily dosing, preferred for: brain oedema, COVID-19 (RECOVERY trial), antenatal lung maturation, croup, antiemesis (chemotherapy), and acute spinal cord compression.
How should long-term steroids be tapered?
Prolonged steroid use (>3 weeks at >5 mg prednisolone/day) suppresses the HPA axis — abrupt withdrawal can cause adrenal crisis (hypotension, hyponatraemia, hypoglycaemia, death). Taper by 10–20% every 1–2 weeks until physiological dose (prednisolone 5–7.5 mg/day), then slower taper. Physiological steroid dose is approximately prednisolone 5 mg/day (equivalent to cortisol production of 15–20 mg/day). Check morning cortisol before stopping completely.
What are the side effects of long-term steroid use?
Metabolic: hyperglycaemia (steroid-induced DM), hyperlipidaemia, weight gain, moon face, buffalo hump, central obesity. Musculoskeletal: osteoporosis (DEXA scan + bisphosphonate prophylaxis if >3 months use), myopathy (proximal weakness — characteristic). Cardiovascular: hypertension, fluid retention. GI: peptic ulcer disease (especially with NSAIDs — PPI prophylaxis). Ophthalmic: posterior subcapsular cataracts, raised IOP/glaucoma. Endocrine: HPA suppression, growth retardation in children. Infection: increased susceptibility, reactivation of TB/strongyloides.
When are steroids used in critical care?
Evidence-based ICU indications: septic shock refractory to vasopressors (hydrocortisone 200 mg/day IV — APROCCHSS trial), ARDS (dexamethasone 20 mg/day × 5 days then 10 mg × 5 days — DEXA-ARDS trial), COVID-19 requiring O2/ventilation (dexamethasone 6 mg/day × 10 days — RECOVERY trial), meningitis (dexamethasone 0.15 mg/kg QID × 4 days — reduces hearing loss in pneumococcal meningitis), anaphylaxis (hydrocortisone 200 mg IV), and acute spinal cord injury (controversial).
What steroid is used for antenatal lung maturation?
Betamethasone 12 mg IM × 2 doses 24 hours apart (or dexamethasone 6 mg IM × 4 doses 12 hours apart) given to the mother at 24–34 weeks gestation when preterm delivery is anticipated within 7 days. Reduces neonatal RDS, IVH, necrotising enterocolitis, and neonatal mortality by 30–50%. A single course is recommended — repeated courses (>2) are associated with reduced fetal growth and neurodevelopmental risk.
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⚠ Critical Safety Warning: Steroid dose equivalences are approximate and based on anti-inflammatory potency only. They do NOT account for differences in route of administration, pharmacokinetics, mineralocorticoid activity, or individual patient factors (hepatic/renal impairment). Do NOT convert steroid doses in patients with adrenal insufficiency using this table — physiological replacement dosing is different from anti-inflammatory dosing. Always verify with a pharmacist or senior physician before switching steroids in critically ill patients.